One in three Americans have a silent blood glucose problem known as insulin resistance. Left untreated, insulin resistance is the cause of many diseases that are actually preventable; such as prediabetes, type 2 diabetes, obesity, heart disease, Alzheimer’s, and cancers.
As insulin resistance develops, the body fights back by producing more insulin. Over months and years, the beta cells in the pancreas that are working so hard to make insulin get worn out and can no longer keep pace with the demand for more and more insulin. (As insulin resistance develops, the body fights back by producing more insulin. Over months and years, the beta cells in the pancreas that are working overtime to make insulin get worn out and can’t keep pace with the demand for insulin. ) This inflammatory response makes it difficult for insulin to communicate with the cells in the liver, muscles, and adipose tissue.
There are several factors that play a role in insulin resistance, but cellular inflammation is the main cause. Cellular inflammation results from an imbalance of two key fatty acids in our blood, Arachidonic Acid (AA) and Eicosapentaenoic Acid (EPA). High levels of arachidonic acid release pro—inflammatory hormones.
Your genetics and ethnic background can increase your risk for developing insulin sensitivity. Driving factors behind insulin resistance are excess body weight, too much belly fat, lack of exercise, smoking, stress, and not enough sleep. Fortunately, you can change these factors. Dietary factors over the long term lead to increased cellular inflammation, making it more likely that you will become insulin resistant. Such as those listed below:
It is difficult to lose weight when we have insulin resistance and cellular inflammation. When we choose the right foods, have the right balance of protein to carbohydrate at each meal, and enough omega-3 fatty acids and polyphenols we can then keep insulin levels in a healthy range; making weight loss and disease prevention easier.
Type 1 Diabetes (formerly called juvenile-onset or insulin-dependent)
In type 1 diabetes, the immune system attacks the cells in the pancreas that make insulin, a hormone that enables the body to use glucose found in foods for energy. As a result, the body does not produce insulin; people with this condition must take insulin daily by injections or an insulin pump. Type 1 diabetes is an autoimmune disorder and the diagnosis occurs in in childhood or young adults. There is no known way to prevent type 1 diabetes, and there is no cure. In adults, type 1 diabetes accounts for approximately 5% of all diagnosed cases of diabetes.
An estimated 1 in 3 U.S. adults age 20 and older have pre-diabetes and the CDC predicts 15% to 30% of people with prediabetes will develop type 2 diabetes within 5 years. An alarming number of people with prediabetes referred for education decline. Many insurance policies do not cover education for prediabetes; most do once a diagnosis of diabetes is made. But don’t wait until then. Prediabetes can be a “symptomless” preview of declining health & wellness or a path to preventing or delaying type 2 diabetes. When the level of (glucose in the blood rises it causes damages to the small blood vessels in the body, and to the heart even in prediabetes.
Risk factors besides being overweight or obese or being age 45 or older are:
Type 2 (formerly called adult-onset or non-insulin-dependent)
Type 2 diabetes (T2D) is the most common form of diabetes; accounting for approximately 95% of cases. It is a metabolic disease involving hormones such as cortisol, leptin, glucagon, and insulin. Leptin regulates blood sugar and helps control appetite as well as fat storage. Leptin also tells the liver what to do with its stored glucose. Insulin is secreted by the beta cells (a type of islet cell) of the pancreas and released when blood glucose is high. The pancreas normally produces enough insulin to handle blood glucose after food is eaten. Insulin is key in releasing blood glucose into the cells where it is converted to energy. Individuals with T2D produce insulin, but their bodies don’t use it correctly; which causes insulin resistant. When someone has insulin resistance the cells do not respond to the insulin as well; and blood glucose cannot enter the cells as easily to produce energy. Because of this, excess glucose builds up in the bloodstream causing type 2 diabetes.
T2D usually occurs in people who are over 40, overweight, have a family history of diabetes, or women who had gestational diabetes. However, T2D is on the rise for adolescents.
Diabetes is an Epidemic
The longer you have insulin resistance, the more harmful diabetes is to the body.
Our expertise helps you to reduce your insulin resistant and live a healthier and happier life.
Type 2 diabetes increases the risk for certain types of cancer, according to a consensus report from the American Diabetes Association and the American Cancer Society. Diabetes doubles the risk for developing liver, pancreatic, or endometrial cancer. Certain medications used for treating type 2 diabetes may possibly increase the risk for some types of cancers.
Diabetes and cancer are common diseases with tremendous impact on health. It appears that they also have commonality in risk factors. According to a consensus report from the American Diabetes Association and the American Cancer Society people who have type 2 diabetes are at greater risk for developing cancer. It states that having Type 2 Diabetes doubles the risk for developing liver, pancreatic, or endometrial cancer; it also increases the risk of colorectal, breast, and bladder cancer by 20% to 50%. People with diabetes tend to have risk factors for cancer such as older age, obesity, poor diet, and physical inactivity.
Type 2 diabetes, obesity, and cancer are characterized by chronic inflammation, which increases production of free radicals that can disrupt insulin signaling and damage DNA. Ensuing genetic mutations can lead to cancer. Adipose cells (fat cells) produce a range of pro-inflammatory cytokines (cell-to-cell signaling proteins), including interleukin-6 (IL-6) and tumor necrosis factor-α (TNF-α). Human studies link elevated levels of IL-6, TNF-α, and C-reactive protein, a biomarker of inflammation, to greater risks for certain cancers and possibly aggressive progression of cancer.
It is important to understand that body fat is a metabolically active; and too much of it can increase cancer risk in the following ways:
Greater body fat, particularly with insulin resistance, tends to increase leptin production. Rising leptin levels further increase hyperinsulinemia, promote inflammation, and induce enzymes that raise estrogen production in postmenopausal women. In cell studies, leptin also directly promotes cell proliferation and angiogenesis and inhibits cell death. Some studies link elevated leptin to increased incidence of colorectal, postmenopausal breast, and other type cancers.
The link between diabetes and certain cancers is well-founded. We know if we reduce obesity and chronic inflammation we can then reduce diseases such as diabetes and cancer.
Children born to women who gained excessive amounts of weight during pregnancy or who struggled with obesity prior to pregnancy are three times more likely to be obese than children whose mothers gained a healthy amount of weight.
Childhood obesity is a growing epidemic in the United States. It affects more than 18 percent of children, making it the most common chronic disease of childhood. This number has more than tripled since 1980. The risk for childhood obesity can actually begin before birth.
We know that metabolic changes that will likely shorten a person’s life span are the result of the increased inflammation in the womb. Therefore, the best way to generate a better metabolic future for the child is to reduce its exposure to its mother’s diet-induced inflammation during pregnancy. If left untreated, woman during pregnancy are a higher risk for metabolic syndrome, gestational diabetes, and eventually diabetes later in life. The driver for this negative fetal programming appears to be increased inflammation in the fetus. Expectant mothers will have a healthier baby by following an anti-inflammatory diet and supplementing with high grade ultra-refined fish oil low in PCBs.
Infants born with high adiposity at birth are more likely to develop cardiovascular and metabolic disease later in life. These outcomes are believed to be associated with an altered lipid exposure in utero. Several papers published the results of a study in which overweight or obese pregnant women were given 2 grams of supplemental EPA and DHA per day starting on week 14 of their pregnancies and continuing until the end of their pregnancies approximately 25 weeks later. Those who got the fish oil had less inflammation as indicated by a reduction of the levels of C-reactive protein (a marker of inflammation). A reduction of inflammation in the womb reduces metabolic problems as the child matures. During pregnancy, omega-3s are a critical building block of the fetal brain, eyes, and nervous system. Omega-3 fatty acids may be the most important nutrients that any pregnant woman can possibly take to reduce inflammation for both mommy and baby.